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...e carried around, but has also been shown to sustain sarcopenia due to a [[macrophage]] infiltration mediated-release of pro-inflammatory [[cytokines]] (such as

...]]. When activated, it binds, with the antigen, to a surface receptor on [[macrophage]]s and [[opsonins|opsonize]] the threatening cells.

...f "killer cells" such as [[lymphocyte#T8 lymphocyte|T8 lymphocytes]] and [[macrophage]]s.

...rface CD47-like virulence factor that contributes to the downregulation of macrophage activation in vivo. Virology , 2005 (Vol. 337)(No. 1) 55-67</ref> ...rface CD47-like virulence factor that contributes to the downregulation of macrophage activation in vivo. Virology, 2005 (Vol. 337)(No. 1) 55-67

...y we deal initially with distributed denial of service and a nonspecific [[macrophage]] response, while we divert the suspicious flow to analysis and start to de

...portin expression in intestinal epithelial cells and reticuloendothelial [[macrophage]]s. As a result, this causes increased levels of serum iron, first biochem

...to diabetes. ''Nature'' 409:307-312</ref><ref>Qatanani M ''et al.'' (2009) Macrophage-derived human resistin exacerbates adipose tissue inflammation and insulin ...’ CD8), which in turn regulate (or fail to regulate) the infiltration of [[macrophage]]s. This permeation of macrophages and their production of proinflammatory

...timulates antibody production, enhances phagocytosis (a process in which a macrophage cell engulfs foreign invaders), and modifies production of cytokines which

...was nearly a hundred times less frequent in endothelial cells compared to macrophage cells. The primary reason being ‘‘L. pneumophila’’ find it easier t

CD8+ effector T cells contribute to macrophage recruitment and adipose tissue inflammation in obesity

...ncy, ultimately leading to adverse long-term outcomes. For example, excess macrophage accumulation and the onset of insulin resistance occur in an adipose tissue ...ses in mass after birth, paralleled by a rise in glucocorticoid action and macrophage accumulation. This process can be reset by changes in the maternal nutritio

** γ3 - expressed in [[macrophage]]s, [[colon (anatomy)|large intestine]], white [[adipose tissue]].

...man)|ovaries]] and [[teste|testes]]. Hardest hit are [[liver]] cells and [[macrophage|macrophages]], or the cells of the [[reticuloendothelial system]]. Lesions

...RANTES (regulated upon activation, normal T-cell expressed and secreted), macrophage inflammatory protein (MIP)–K have been detected in the nasal secretions.

...acrophages]], which are unable to kill the bacteria. The bacteria kill the macrophage and migrate to the [[lymph nodes]] where they are phagocytosed by the [[pol

...way that regulates defense against oxidative stress, which may impair lung macrophage function. In both COPD patients and a mouse model of COPD, a phytochemical

* Poor iron reutilization from macrophage recirculation in the [[bone marrow]]

...ee illustration) that lives within vacuoles of [[protist]] and mammalian [[macrophage]] cells.<ref>Jain R ''et al.'' (2003) Horizontal gene transfer accelerates

*The timing of TNF and IFN-γ signaling affects macrophage activation strategies during Mycobacterium [[tuberculosis]] infection

...ee illustration) that lives within vacuoles of [[protist]] and mammalian [[macrophage]] cells.<ref>Jain R ''et al.'' (2003) Horizontal gene transfer accelerates