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...bly as part of infection with a pathogen). These cells are recognised by [[macrophage]]s or [[dendritic cell]]s which act as antigen presenting cells, this cause ...kin-4 and interleukin-5, which promote B-cell production of IgE and IgG4, macrophage deactivation, and mast-cell and eosinophil responses. Rashes that are media

...[[monocyte]]s, [[neutrophil|polymorphonuclear neutrophils]], and tissue [[macrophage]]s of the spleen and liver. The latter are sometimes called part of the [[r

...cytokine]]s, [[chemokine]]s and lysosomal enzymes, from [[neutrophil]]s, [[macrophage]]s and [[mastocyte]]s.

Most iron is recycled, principally by [[macrophage]]s in the [[reticuloendothelial system]]. ...f [[interleukin|interleukin-6 (IL-6)]]. Increased hepcidin traps iron in [[macrophage]]s, decreases plasma iron concentrations, and is a mechanism of [[anemia of

...ic use, but current thinking is that they decrease antigen production by [[macrophage]]s and [[lymphoid dendritic cell]]s. They do this by increasing their intra

...shown that the clinical strain was very effective in inducing apoptosis in macrophage while the environmental strain was not able to. ...at neutrophils alone cannot prevent the infection but rather only help the macrophage in inhibiting the growth of this bacterium.<ref>[http://www.pubmedcentral.n

...form committed stem cells (i.e., progenitors). The cytokine [[granulocyte-macrophage colony-stimulating factor]] ([[GM-CSF]]) causes differentiation into eosino

'''Granulocyte-macrophage colony-stimulating factor (GM-CSF)'''<ref>Some papers spell it "granulyte".

*Possible suppression of macrophage involvement of prostaglandin synthesis and modulation of [[interleukin-10]]

TNF is a [[cytokine]] produced by activated [[macrophage]]s, and plays important roles in both apoptotic and cell survival processes

...tosis]], agglutination as by [[eosinophil]]s, nonspecific recognition by [[macrophage]]s

...e carried around, but has also been shown to sustain sarcopenia due to a [[macrophage]] infiltration mediated-release of pro-inflammatory [[cytokines]] (such as

...]]. When activated, it binds, with the antigen, to a surface receptor on [[macrophage]]s and [[opsonins|opsonize]] the threatening cells.

...f "killer cells" such as [[lymphocyte#T8 lymphocyte|T8 lymphocytes]] and [[macrophage]]s.

...rface CD47-like virulence factor that contributes to the downregulation of macrophage activation in vivo. Virology , 2005 (Vol. 337)(No. 1) 55-67</ref> ...rface CD47-like virulence factor that contributes to the downregulation of macrophage activation in vivo. Virology, 2005 (Vol. 337)(No. 1) 55-67

...to diabetes. ''Nature'' 409:307-312</ref><ref>Qatanani M ''et al.'' (2009) Macrophage-derived human resistin exacerbates adipose tissue inflammation and insulin ...’ CD8), which in turn regulate (or fail to regulate) the infiltration of [[macrophage]]s. This permeation of macrophages and their production of proinflammatory

...timulates antibody production, enhances phagocytosis (a process in which a macrophage cell engulfs foreign invaders), and modifies production of cytokines which

...was nearly a hundred times less frequent in endothelial cells compared to macrophage cells. The primary reason being ‘‘L. pneumophila’’ find it easier t

...ncy, ultimately leading to adverse long-term outcomes. For example, excess macrophage accumulation and the onset of insulin resistance occur in an adipose tissue ...ses in mass after birth, paralleled by a rise in glucocorticoid action and macrophage accumulation. This process can be reset by changes in the maternal nutritio

...man)|ovaries]] and [[teste|testes]]. Hardest hit are [[liver]] cells and [[macrophage|macrophages]], or the cells of the [[reticuloendothelial system]]. Lesions