Insulin resistance: Difference between revisions
Jump to navigation
Jump to search
imported>Gareth Leng No edit summary |
imported>Anthony.Sebastian (add pre-note & link) |
||
Line 1: | Line 1: | ||
{{subpages}} | {{subpages}} | ||
:See: ''Article on [[insulin]], if necessary, to better understand present article.'' | |||
In [[medicine]], '''insulin resistance''' is "diminished effectiveness of [[insulin]] in lowering blood sugar levels: requiring the use of 200 units or more of insulin per day to prevent hyperglycemia or ketosis. Tt can be caused by the presence of insulin antibodies or the abnormalities in insulin receptors (receptor, insulin) on target cell surfaces. It is often associated with [[obesity]]; [[diabetic ketoacidosis]]; [[infection]]; and certain rare conditions."<ref>{{MeSH}}</ref> | In [[medicine]], '''insulin resistance''' is "diminished effectiveness of [[insulin]] in lowering blood sugar levels: requiring the use of 200 units or more of insulin per day to prevent hyperglycemia or ketosis. Tt can be caused by the presence of insulin antibodies or the abnormalities in insulin receptors (receptor, insulin) on target cell surfaces. It is often associated with [[obesity]]; [[diabetic ketoacidosis]]; [[infection]]; and certain rare conditions."<ref>{{MeSH}}</ref> | ||
Revision as of 19:01, 7 November 2012
![](http://s9.addthis.com/button1-share.gif)
- See: Article on insulin, if necessary, to better understand present article.
In medicine, insulin resistance is "diminished effectiveness of insulin in lowering blood sugar levels: requiring the use of 200 units or more of insulin per day to prevent hyperglycemia or ketosis. Tt can be caused by the presence of insulin antibodies or the abnormalities in insulin receptors (receptor, insulin) on target cell surfaces. It is often associated with obesity; diabetic ketoacidosis; infection; and certain rare conditions."[1]
Insulin resistance (HOMA-IR) can be measured by:[2]
References
- ↑ Anonymous (2024), Insulin resistance (English). Medical Subject Headings. U.S. National Library of Medicine.
- ↑ Matthews DR, Hosker JP, Rudenski AS, Naylor BA, Treacher DF, Turner RC (1985). "Homeostasis model assessment: insulin resistance and beta-cell function from fasting plasma glucose and insulin concentrations in man". Diabetologia 28: 412–9. PMID 3899825. [e]