User:Anthony.Sebastian/Saved References: Difference between revisions
Jump to navigation
Jump to search
imported>Anthony.Sebastian (Created page with "<!-- Please ignore (but don't delete) any formatting that you are not familiar with. Others will probably chime in to help you set things up. Start your new article by replacing ...") |
No edit summary |
||
(One intermediate revision by one other user not shown) | |||
Line 1: | Line 1: | ||
{{AccountNotLive}} | |||
<!-- Please ignore (but don't delete) any formatting that you are not familiar with. Others will probably chime in to help you set things up. Start your new article by replacing these lines! If it is your first one, you may have a look at [[CZ:Quick Start]], and if you cannot find it, just press the "Save page" button below this edit window — it will then be linked from here.--> | <!-- Please ignore (but don't delete) any formatting that you are not familiar with. Others will probably chime in to help you set things up. Start your new article by replacing these lines! If it is your first one, you may have a look at [[CZ:Quick Start]], and if you cannot find it, just press the "Save page" button below this edit window — it will then be linked from here.--> | ||
Line 6: | Line 7: | ||
==References== | ==References== | ||
<references/> | <references/> | ||
Latest revision as of 02:52, 22 November 2023
The account of this former contributor was not re-activated after the server upgrade of March 2022.
- Mitochondrial response to controlled nutrition in health and disease[1]
References
- ↑ Schiff M, Benit P, Coulibaly A, Loublier S, El-Khoury R, Rustin P. (2011) Mitochondrial response to controlled nutrition in health and disease. Nutr Rev 69:65-75.
- Abstract: Mitochondria exert crucial physiological functions that create complex links among nutrition, health, and disease. While mitochondrial dysfunction with subsequent impairment of oxidative phosphorylation (OXPHOS) is the hallmark of the rare inherited OXPHOS diseases, OXPHOS dysfunction also plays a central role in the pathophysiology of common conditions such as type 2 diabetes and various neurodegenerative disorders. Dietary interventions, especially calorie restriction, have been shown to improve the course of these diseases and to extend the lifespan. Few data are available on the impact of nutraceuticals (macronutrients, vitamins, and cofactors) on primary inherited OXPHOS diseases. This review presents recent knowledge about the impact of nutritional modulation on mitochondria and lifespan regulation and about the development of potential treatments for mitochondrial dysfunction diseases.